More about Nicotine and the spike protein (important info)

paper regarding the binding of S1 Spike to acetylcholine receptors.
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The majority of adverse reactions to the V are neurological - and it is becoming extremely obvious that this is the main route of attack of the V upon the nervous system i.e via the nACh receptors.
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This paper provides some fascinating observations regarding the effect of nicotine.
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It also identifies the exact sequence of aminoacids within the Spike that are responsible for the binding to nACh receptors.
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This is the sequence - YQTQTNSPRRAR
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This sequence is very similar to sequences from snake neuro-toxins and from Rabies sequences that also operate by binding strongly to acetylcholine receptors, and consequently are also highly neuro-toxic. In the comments below I have included these sequences for comparison.
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It seem that the part of the S1 Spike that has affinity for the acetylcholine (nACh) receptors is the Furin Cleavage site PRRA, the very site which is missing from all other "C 0r-0n@-v1r-u$e$" - hence most likely to have been added by gain-of-function engineering.
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Based on these observations, it would seem that nicotine, by binding to acetylcholine receptors in preference to the Spike, will greatly reduce the incidence of neurological pathologies following the V.
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Interestingly, "Ivor Makes Tin", which has been recently shown to inhibit the replication of SARS-CoV-2 in cells in vitro [53], is a positive allosteric modulator of nAChR .
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R. M. Krause; B. Buisson; S. Bertrand; P. J. Corringer; J. L. Galzi; J. P. Changeux; D. Bertrand Ivor make tin: a positive allosteric effector of the alpha7 neuronal nicotinic acetylcholine receptor, MolPharmacol, Volume 53 (1998), pp. 283-294
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[link to comptes-rendus.academie-sciences.fr (secure)]